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Consequently, this suggests that all of the emphasized failure points (i.e., failed blood flow, osteoblast proliferation, and muscle tension) should be resolved, so that muscle-to-bone healing can be ongoing [18]

The mechanism involves angiogenesis (new blood vessel formation) and upregulation of growth factor receptors at the injury site

Rescue medication was available for patients whose fasting plasma glucose exceeded the predefined limits of 15.0 mmol/L from week 8, 13.3 mmol/L from week 9 to week 20, and 11.1 mmol/L from week 21 to week 32

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Cagrilintide is engineered around that problem: it is a modified, lipidated peptide that acts as a dual amylin and calcitonin receptor agonist and is stable enough for once-weekly subcutaneous dosing